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Through controlling these targets, HSF1 acts in diverse stress-induced cellular processes and molecular mechanisms, including the endoplasmic reticulum unfolded protein response and ubiquitinproteasome system, multidrug resistance, autophagy, apoptosis, immune response, cell growth arrest, differentiation underlying developmental diapause, chromatin remodelling, cancer development, and ageing

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POD activities significantly decreased compared to those not before treatment but treated with 1 and 2 C (Fig

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The acute withdrawal symptoms generally begin to subside significantly after the first month, though some symptoms may persist at reduced intensity

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