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The fate of serine metabolism on macrophage polarization was investigated and a novel mechanism was proposed by which serine deficiency stimulates the M1 phenotype and inhibits STAT6-mediated M2 polarization by activating p38-dependent JAKSTAT-1 axis [286], suggesting that serine metabolism may be an underlying target

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Similarly, arginine-rich diets or supplementation can counteract arginine depletion by MDSCs, enhancing T cell proliferation and function [132]

Safety Measurements Any adverse event during the course of the study was monitored and reported to the study staff at week 6 (visit 5) or week 12 (visit 8) in the clinic or directly to the principal investigator throughout the study

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