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doi: 10.1146/annurev-arplant-081320-092855

Fasn inhibition was shown to reduce endothelial damage and functional changes by suppressing oxidative stress and NLRP3 inflammasome-dependent pyroptosis through activation of the Nrf2/HO-1 pathway

Fibroblasts are generally quiescent under normal physiological conditions but are activated during tumorigenesis via complex signaling pathways to support tumor growth and progression [21,22]