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Possibilities for therapies targeting cholesterol uptake in breast and ovarian cancers Cancers often upregulate the key cholesterol uptake molecules such as NPC1, NPC1-like 1 protein (NPC1L1), LDLR, SR-B1, and proprotein convertase subtilisin/kexin like 9 (PCSK9) making them the most logical targets of possible pharmaceutical interventions

While mRNA levels may not directly reflect protein presence, this finding argues against a potential simple explanation for DTT resistance in our mutants, whereby rips-1 mutation results in the loss of DTT uptake

Mbio (2017) 8:e02183-16

Quantification and statistical analysis Qualitative data were shown as frequency with percentage, while quantitative data were shown as median with interquartile range

It also reduced the number of activated microglia and astrocytes around amyloid plaques, thereby reducing neuroinflammation

Gohda, T