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Energy expenditure (possible): Some evidence amylin may increase energy expenditure Not as strong as mechanism Minor contributor if real Why amylin is effective for obesity Physiologic rationale: Amylin naturally controls meal size Obesity often associated with amylin resistance Type 2 diabetics have reduced amylin Replacing/supplementing amylin restores control Addresses root cause Synergy with insulin: Both co-secreted normally Work together to regulate feeding Amylin prevents overeating Insulin handles glucose Complementary hormones Comparison to GLP-1 mechanisms: GLP-1: Moderate gastric slowing, strong central appetite effects Amylin: Very strong gastric slowing, moderate central effects Different receptor pathways Complementary not redundant Can combine for synergy Get personalized weight loss protocols at SeekPeptides using our peptide calculator and peptide cost calculator

doi:10.1021/acschemneuro.5b00031

The theoretical advantage is that synaptogenesis and neuroplasticity are supported through independent signaling cascades, potentially producing greater effects than either pathway alone

MPTP selectively destroys dopaminergic neurons in the substantia nigra, producing a pattern of damage that closely mirrors Parkinson pathology

Kang, Y.-H

Phase III is for fielding