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Based on the increase in cardiolipin biosynthesis, we hypothesize that luseogliflozin facilitates the maintenance of mitochondrial ATP production in response to I/R injury-induced tissue damage and oxidative stress

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The data were fitted to a nonlinear substrate inhibition model ( y = V max x /( K m + x (1 + x / K i ))

10 l from each well was acquired on a BD FACSCelesta, then the median fluorescence intensity (MFI) of the PE-positive cells was calculated for each well using FlowJo v10.8 Software (BD Life Sciences)

Tissues were homogenised in a cooled Tissuelyser (Bertin Technology), with 7200 rpm, 20 s of shaking separated with a 20-s break