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Orphan nuclear receptor ERR controls macrophage metabolic signaling and A20 expression to negatively regulate TLR-induced inflammation

This sequence of events is recapitulated in a humanized mouse model of diabetes, in which the presence of autoreactive T-cells alone was insufficient to induce disease, which was triggered only when -cells were stressed by the addition of the diabetogenic agent, streptozotocin (STZ) (32)

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Extracted DNA was used for downstream Illumina sequencing

Earlier diagnosis of peripheral neuropathy in primary care: a call to action