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Nietzel et al., 2020) whereas CysC (Cys150) could be sulfenylated (Wei et al., 2020)

This allows researchers to assess how glutathionylation fluctuates in response to oxidative stimuli, metabolic shifts, or genetic perturbations

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This is particularly relevant in tumor cells, as described below

High glucose-induced glutamine sparing triggers RUNX2 loss under diabetic conditions Under high-glucose conditions, mTORC1 is hyperactivated, which results in feedback inhibition of mTORC2 through p-70S6K and finally a loss of RUNX2 protein levels

Front Cell Dev Biol (2021) 9:748925