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GSH administration effectively reversed this downregulation of SOD (Figure 5D)

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The process of NETosis mediated by reactive oxygen species (ROS)-derived from mitochondria and NADPH oxidase could induce autoantibody production, resulting in uncontrolled inflammatory response and tissue pathology [88]

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Sat1 inhibition resulted in the reversal of increased lipid peroxidation levels, as shown by the results of flow cytometry (Figures 4H, I)

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