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To cope with the elevated oxidative stress they generate, cancer cells enhance their antioxidant capacity, largely through sustained activation of the transcription factor NRF2 often driven by mutations in its negative regulator KEAP1

A rare but clinically significant manifestation is pseudo-thrombotic microangiopathy (PTMA), which closely resembles true thrombotic thrombocytopenic purpura (TTP), characterized by hemolytic anemia, thrombocytopenia, the presence of schistocytes, and elevated lactate dehydrogenase (LDH) levels [2]

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The mechanisms are different and do not directly conflict, but absence of conflict is not the same as evidence of benefit

Selenium in the testis of the rat: studies on its regulation and its importance for the organism