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For instance, it has been recently observed that in prostate cancer patients, polymorphism in GSTM3-3 may contribute to resistance to hormonal therapy through oxidative stress 45

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This growth was driven by the identification of the immunosuppressive 4 and anticancer properties of rapamycin, 5 as well as the discovery of its molecular target, a protein named target of rapamycin (TOR), first in yeast 6,7 and three years later, its homolog in mammals, subsequently referred to as mammalian TOR (mTOR)

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Defective mitochondrial morphology and bioenergetic function in mice lacking the transcription factor yin yang 1 in skeletal muscle

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