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discovered that ALDOA promotes tumor glucose metabolism and growth, and that HDPS-4II, by specifically inhibiting ALDOA, significantly suppresses glucose metabolism and tumor growth in HCC, highlighting its potential as a novel therapeutic approach for HCC (214)

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Several genes were differentially expressed with GO treatment: 104 differentially expressed genes were shared between the 15 min and 1-h timepoints and 43 genes were unique to the 1-h or late expressed timepoint

Tissue-specific genetic models, single-cell transcriptomics, and proteomic approaches will be critical for defining these pathways

Torrens-Spence et al