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The peer-reviewed literature on GHRP-class cytoprotection encompasses GHRP-2 as a high-potency member of this pharmacological framework, with cardioprotective studies specifically documenting CD36-dependent mechanisms consistent across the GHRP-2, GHRP-6, and Hexarelin subclass

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High concentrations and prolonged exposure to ROS causes extensive damage to various integral cellular biomolecules (Figure 1B), including proteins, lipids and nucleic acids, which ultimately hampers multiple cellular functions (49, 88)

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