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doi: 10.1111/head.13808 207

Body composition changes are clearly evident for most researchers following consistent protocols with proper diet and training

Our findings delineate a novel mechanism of communication from mitochondria to the ER, reveal an unexpected protective role for GSSG, and provide a plausible pathway by which ER homeostasis is linked to metabolic activity

Subclinical myocarditis can produce microscopic fibrosis, which infection later amplifies, increasing susceptibility to arrhythmias, contractile dysfunction, and heart failure

Inhibition of USP1 activates ER stress through Ubi-protein aggregation to induce autophagy and apoptosis in HCC

These studies thus support that mTOR signaling represents a potential therapeutic target for fragile X syndrome and other ASDs