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The catalytic and anti-ferroptotic mechanisms of FSP1 Like NDH-2 enzymes, FSP1 could form a ternary complex after binding NAD(P)H and ubiquinone at two different sites (Fig

A: Unlike NSAIDs, BPC-157 does not appear to damage the GI tract

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It appears to function primarily in the absorption of dietary short-chain monocarboxylates rather than those produced by bacterial fermentation [80]

265 Taken together, regulating chemokine expression, especially the CCL2/CCR2 signaling pathway, may serve as a potential therapeutic approach against cerebral ischemic stroke, although the harmful effects of such an intervention should be carefully considered