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Combined E1A gene therapy and taxol in HER-2/neu-overexpressing ovarian cancer: E1A chemosensitization through apoptosis involving caspase-3 pathway

FbpA, ferric-binding protein

This mechanism involves a combination of oxidative stress and the formation of iron-dependent lipophilic ROS, e.g., lipid hydroperoxides

Physiological assessment of heat tolerance Leaf EL was analyzed to evaluate cell membrane stability

Other than the conventional H-bonds, carmustine also interacts with residues in close proximity by van der Waals interactions with residues Pro160, Thr176, Ser177, Gly194, Ala195, Trp287, Ile289, Gly290