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However, it was not recognized at the time that evidence for the protein binding hypothesis was obtained in animals on a normal diet whereas the LPO hypothesis was derived from animals fed a vitamin E-deficient diet high in polyunsaturated fatty acids (PUFAs), i.e., in animals highly susceptibility to LPO [61]

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however, Mn can also instigate autophagic flux and dysfunction, especially under conditions of low Mn levels [29]

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Additionally, oxPTM-INS-Ab were detectable prior to clinical onset, identifying children at risk of progressing to clinical T1D (94)

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