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Dear editor A recent study published in Annals of the Rheumatic Diseases suggests that the expression of hypoxia-inducible factor 1 alpha (HIF1a) in intestinal epithelial cells (IECs) represents a critical check point for the development of arthritis by inhibiting necroptosis of IECs and preserving the intestinal barrier integrity [1]

Figure 9a and b show the MichaelisMenten plots for HO and TMB substrates, respectively, while Fig

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Prevalence of SDHB, SDHC, and SDHD germline mutations in clinic patients with head and neck paragangliomas

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