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Are nutrient and protein oxidation inherently incompatible due to redox limitations

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Building on the redox-linker strategies described earlier, scaffold design leverages disulfide- and boronate-based networks to achieve tunable degradation and on-demand GSH release

1 The exportin-1 inhibitor selinexor was evaluated in the phase 3 BOSTON study of selinexor, bortezomib, and dexamethasone therapy administered once weekly versus twice weekly doublet therapy with bortezomib and dexamethasone

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These perturbations lead to the release of mtDAMPs, rewire immune cell metabolism toward pro-inflammatory or immunosuppressive phenotypes, and impair the bioenergetic capacity needed for effective host defense and tissue repair ( Figure 2 )