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Although the above results indicated that essential cytosolic disulfide reducing power in TrxR1/Gsr-null livers was coming from newly synthesized GSH, it was unclear how the hepatocytes could acquire the Cys used to synthesize GSH

Sources: Klihada, F

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Clinical pathobiochemistry of vitamin B12 deficiency: improving our understanding by exploring novel mechanisms with a focus on diabetic neuropathy

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