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This switch is controlled by TRPM7-dependent Ca 2+ influx and requires actin polymerization, myosin recruitment and the formation of focal adhesions at the interface between the cell and the lateral channel walls

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Asked recently whether the molecule is even what the original team believed it was, one of Sikirics collaborators told Undark, As long as it works and has no side effects, who cares? If you read my work, you know Im not big for pathophysiology