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albicans , and eventually exerting antifungal activities [153,154,155,156]

However because of methodological difficulties the data does not allow for a very confident prediction

All considered, these data strongly suggest that SAMe depletion contributes to multiple pathophysiological processes observed in the AD brain, including the unsilencing of genes involved in AD development and progression, the depletion of acetylcholine, and the inability to appropriately dephosphorylate tau

Arch Toxicol, 55: 186-190

Normally, glutathione helps neutralize NAPQI, but in an overdose, glutathione stores can become depleted