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409,410,411 In addition, molecular mimicry between EBV proteins and autoantigens provides another possible model for disease induction and immune escape

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HFD can metabolic dysfunction-associated steatotic liver disease (MASLD) by increasing the uptake of free fatty acids by hepatocytes, disrupting the balance between de novo synthesis and oxidative degradation of fatty acids, leading to excessive deposition of triglycerides (TG) in hepatocytes