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In: akatay U (ed) Redox signaling and biomarkers in ageing

In contrast, the opposite phenotype of lipid peroxidation, iron imbalance, and mitochondrial structural damage was observed after the silencing of HIF-1 (Fig

A novel quinoline derivative, DFIQ, sensitizes NSCLC cells to ferroptosis by promoting oxidative stress accompanied by autophagic dysfunction and mitochondrial damage