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One explanation might be that the roles of GH and GHR on mitochondrial biogenesis may be different among cell-, organ-, and species-specific factors

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Unresolved inflammation results in formation of vulnerable plaques characterized by enhanced macrophage apoptosis and defective efferocytosis of apoptotic cells resulting in necrotic cell death leading to increased smooth muscle cell death, decreased extracellular matrix production, and collagen degradation by macrophage proteases

This mechanism is similar to how minoxidil works, but copper peptides do so without the risk of irritation or dryness

VEGF, in addition to acting as a mitogenic signal for vascular endothelial cells, protects tumor vasculature from apoptosis by inducing anti-apoptotic factors, mediates the activation of extracellular matrix degrading enzymes, and promotes tumor-immune evasion by recruiting immunosuppressive cells such as Tregs, TAMs, and MDSCs

HGF heptocyte growth factor MSP macrophage stimulating protein the AnglV analog Norleual which is a potent inhibitor of the HGF/Met system, was shown to bind to HGF and block its dimerization (Kawas et al, 2011)