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Mitophagy in cisplatin ototoxicity Although most previous studies have shown nuclear DNA as the target of cisplatin in cochlear cells, recent studies provide the support that mitochondria are also the major sites of cisplatin cytotoxic damage

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Activity of nedocromil sodium in mast-cell-dependent reactions in the rat

Animal studies have shown downregulation of HIF-1 and downstream pro-survival proteins (e.g., glucose transporter 1, vascular epithelial growth factor, and carbonic anhydrase) following administration of oral or parenteral vitamin C ( Figure 1 ) (Kuiper et al., 2010, 2014a

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Journal of Korean Medical Science