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Defects in their distribution or function trigger axonal degeneration prior to somal damage [23]

221 (9), e202112107

Proc Natl Acad Sci USA 93:47604764

It also recycles vitamins C and E back to their active forms and takes part in the liver's normal processing of some compounds

3i): Together with homocysteine, adenosine is the product of SAH hydrolysis by AHCY, and thus a substrate for the reverse reaction when other routes of adenosine metabolism are interrupted, causing an increase in SAH 62,63