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Temporary S-glutathionylation of enzymes in the TCA cycle, ETC, or -oxidation pathway protects them from irreversible oxidation when ROS is high but may also hypothetically serve as a mechanism to diminish mitochondrial ROS production by reducing their activity [51]

3 Every time the brain worksthinking, focusing, processing, rememberingthat activity generates its own byproduct: oxidative stress

Arch Neurol 64(7):938944 Strijks E, Kremer H, Horstink M (1997) Q10 therapy in patients with idiopathic Parkinsons disease

Surprisingly, we also identified an essential, non-canonical function of the glutathione synthesis enzyme GCLC under cystine starvation conditions

Unfortunately, no concrete evidence of their efficacy exists to support their beneficial use in addressing the symptoms of mitochondrial diseases [9, 10]

Borrelia-specific interferon-gamma and interleukin-4 secretion in cerebrospinal fluid and blood during Lyme borreliosis in humans: association with clinical outcome