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In: Autophagy: Cancer, Other Pathologies, Inflammation, Immunity, Infection, and Aging (2015)

Glial cells as regulators of neuroimmune interactions in the central nervous system

When KCF18 was bound to these cytokines, electrostatic interactions gave the dominant contribution to the computed MM/PBSA binding affinities whereas the contribution due to VDW interactions was minor (Supporting Figure S2A)

Preliminary research suggests that these mechanisms may be similar to those observed with exogenous hGH therapy, including the activation of the adrenergic receptors and increased lipolysis in adipocytes [5]

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