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The lack of BH4 may be caused by excessive oxidative stress, which causes BH4 to be oxidized to BH2 without sufficient recycling back to BH4

BLACKWOOD, J.D

Moreover, a reactive quinone methide metabolite of dauricine was observed and identified in MLMs, which spontaneously captured by GSH (Figure 4), and its GSH adducts can be suppressed by CYP3A inhibitor ketoconazole (Xie et al., 2016)

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Methods: Using the UNOS database, a retrospective cohort was extracted of recipients of first liver transplant with renal dysfunction (estimated GFR 2 mg/dL) and the impact of pre-LT MELD, pre-LT renal disease (acute kidney injury [AKI] or chronic kidney disease [CKD]), liver disease etiology (MASH vs alcohol-related liver disease [ALD] and other non-MASH), and 1-year trough tacrolimus (Tac) level on developing renal dysfunction following LT

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