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1 Introduction The pathogenesis of autoimmune type 1 diabetes mellitus (T1D) involves infiltration of the pancreatic islet cells by T-lymphocytes, macrophages, and other immune cells with consequent loss of insulin producing -cells ( Outside of the CNS, GABA is highly concentrated in the pancreatic islet wherein it has autocrine and paracrine actions to regulate -cell insulin secretion and inhibit -cell glucagon release

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S.Sagi-KissV.et al

Therefore, no SLC1A5 inhibitors with high specificity have been identified or designed, and SLC7A11 and GLUD inhibitors have not yet been applied to clinical oncology because of toxicity or efficacy as tool compounds [167, 170]

J Pineal Res 44(4):387396

Immune factors do not act in isolation but form a vicious cycle with other characteristics of PCOS, such as insulin resistance and hyperandrogenism (20)