Notably, pirfenidone therapy has been shown to reduce the Kyn/Trp ratio, suggesting that modulation of the Kyn-Trp pathway may contribute to its antifibrotic effects [142]
Leucine-, arginine-, and methionine-derived SAM can bind their sensors during high intracellular concentrations, thereby disrupting the interaction of the sensors with GATOR1/2, relieving the inhibition of GATOR complex, and activating mTORC1
doi: 10.1007/978-1-4939-0897-4_5 85
In conclusion, affecting the synthesis of ISC by inhibiting NFS1 and thus altering the sensitivity of cells to ferroptosis is a potential therapeutic tool to promote ferroptosis in lung cancer, but the precise mechanism remains to be further investigated
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