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doi: 10.1148/radiology.219.1.r01ap2986

C., Parakh, S., Lee, F

However, the main activator of S473 kinase is thought to be mTORC2, since inactivation of mTORC2 results in a dramatic decrease in S473 phosphorylation [18, 32, 33]

SAG has demonstrated efficacy in replenishing intracellular GSH in cultured fibroblasts from patients with genetic glutathione synthetase deficiency a condition where the de novo synthesis pathway is non-functional