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ROS are counterbalanced by an antioxidant defence network, which functions to modulate ROS levels to allow their physiological roles whilst minimizing the oxidative damage they cause that can contribute to disease development

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There is a variety of animal models of SAH currently available [128], and ex vivo and in vitro studies are utilized to evaluate the effects of SAH, blood products, compounds, etc., on isolated arteries and various brain cell types

High-fat-fed ApoE / mice exhibit larger atherosclerotic plaques, severe ferroptosis, increased NOX production, and reduced GSH and xCT expression in the thoracic aorta compared to wild-type mice ( / mice

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