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As mitochondria participate in intracellular calcium buffering, their dysfunction can lead to intracellular calcium overload, activating calcium-dependent proteases, disrupting cellular structure, and promoting apoptosis

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Following T cell activation, CD154 is expressed on the surface of the cell, allowing binding to B cells through CD40

Reactive astrocytes transduce inflammation in a bloodbrain barrier model through a TNF-STAT3 signaling axis and secretion of alpha 1-antichymotrypsin

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Excessive mtROS and disrupted mitochondrial dynamics amplify inflammation and impair host defense