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Biol Reprod (1998) 59(3):599605

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Research reported in this publication was supported by the Maximizing Investigators Research Award (MIRA R35) from the National Institute of General Medical Sciences (NIGMS) of the National Institutes of Health (NIH) #R35GM138183 to J.R.B

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Beyond its effects on nasal epithelial cells, H1N1 infection also accelerates ferroptosis and lung injury via tripartite motif containing 46 (TRIM46)-mediated ubiquitination of SLC7A11 (Zhou et al., 2024a)

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