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These microbial signatures can be used to not only predict disease risk but also forecast the likelihood of treatment success, offering a new paradigm in precision medicine

These results suggested that in the cellular context, the ASXL1 1-645 cancer-linked truncation variant shows an increased ability to bridge BRD4 to some ASXL1-regulated genes, and the ASXL1 1-591 truncation may be less efficient in linking BRD4

doi: 10.3390/ijms20040905

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