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Despite secreting substantial amounts of Interleukin-17 (IL-17) and TNF-, these cells show a significant reduction in cytotoxic function, which may lead to an increased risk of infection and carcinogenesis [35]

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Therapeutic regimens employing IFN- and TNF- to counteract the immunosuppressive microenvironment of GBM have shown promise in preclinical models and early clinical trials [519521]

Excessive ROS production triggered by ZEA compromises mitochondrial membrane potential, leading to structural and functional damage [49]