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First, the regulatory mechanisms that control the switch between the two viral life cycles are complex and sophisticated

To explore this notion, we installed the BDHI electrophile onto a set of chemical fragments (so called scout fragments) previously shown to drive covalent protein engagement with different electronic and steric properties 2 and assessed their proteomic reactivity

In vivo experiments in mice highlighted that the transplantation of osteosarcoma cells stably overexpressing miR-26a, suppressed tumor growth (76)

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Yeast screen for constitutively active mutant G protein-activated potassium channels