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5.3.3 Progress in drugs targeting A1 astrocytes The transformation of astrocytes into the neurotoxic A1 phenotype following TBI represents a critical factor exacerbating secondary injury

An empirical study on large-scale multi-label text classification including few and zero-shot labels

Reference Mukanganyama, Bezabih, Robert, Ngadjui, Kapche, Ngandeu and Abegaz2011

Additionally, post-operative IF prevented microglial activation and the elevation of sphingosine-1-phosphate receptor 1 and inflammatory cytokines in the hippocampus of 2VO rats

GCDH loss did not alter 2-OG metabolism, nor 2-HG levels

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