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Transient FAPCAR-T cells generated in vivo via the LNP-mRNA system cleared overactivated fibroblasts and have previously been shown to inhibit the development of cardiac fibrosis 12

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This may reduce concerns around cell proliferation and metabolic disruption two of the primary safety concerns with hGH therapy.[1] Emerging Joint and Cartilage Support Beyond fat metabolism, early research points to potential joint health benefits