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The latencies for scores 2 and 3 increased when treated with DZP at 18 M compared to the PTZ group

The modification extends the peptides half life

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In particular, the quantity and timing of carbohydrate, protein and fat consumption are manipulated to optimise substrate availability and training adaptations (Reference Thomas, Erdman and Burke16)

The loss of a targeted mutation has previously been interpreted as evidence for correction and efficient repair between homologous chromosomes at MYBPC3 (12), which appeared to elevate such promise