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Some individuals with inherited deficiencies of specific enzymes that synthesise glutathione may develop neurological complications later in life 22

Glucocorticoid programming in very preterm birth

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Sp could restore the redox balance, mitochondrial function, and prevented oxidative damage, leading to recovery of dopaminergic neural networks and motion abilities in Drosophila genetic model of PD

2.1.3 Calcium dysregulation and excitatory neurotransmission METH enhances glutamate release, leading to NMDAR overactivation and increased calcium influx into neurons (Staszewski and Yamamoto, 2006)

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