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& Albensi, B

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In all, the data demonstrate that supraphysiologic increases in mitochondrial ubidecarenone levels is sufficient to alter redox homeostasis as well as promote site-specific ROS production at the level of the mitochondria at Complex II and mGPDH

Tradit Chin

Abstract Neurotrophin signaling through NGF/TrkA and BDNF/TrkB is increasingly recognized as a driver of osteosarcoma (OS) progression and an organizer of its immune milieu, yet clinical translation has lagged amid intratumoral heterogeneity and a myeloid-skewed, vasculature-aberrant tumor microenvironment (TME)

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