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Abstract Large language models (LLMs) show great potential for clinical decision-making, yet most applications remain narrow, task-specific chat tools rather than systems integrated into clinical workflows 1,2

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In a folic acid-induced animal model of AKI, ferroptosis has been implicated in the development of renal fibrosis [100, 101]

Parasitology (2003) 127:34956

This review examines the molecular mechanisms through which red blood cells adapt to hypoxic conditions, including nitric oxide (NO)-driven changes in membrane properties, Cys93-dependent S-nitrosylation, adenosine-induced activation of glycolysis, and the development of hypoxic memory via eENT1 degradation