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Advanced glycation end products induce skeletal muscle atrophy and insulin resistance via activating ROS-mediated ER stress PERK/FOXO1 signaling

B cells in the pathophysiology of myasthenia gravis

Our previous research showed that hepsin / mice exhibited decreased HGF-cMet signaling, with reduced levels of activated HGF and phosphorylated cMet, resulting in a two-fold increase in GJ expression compared to wild-type mice (Hsu et al

PMID: 41857421 Am J Pathol Hyperglycemia induces tear reduction and dry eye in diabetic mice through the norepinephrine-1AR -mitochondrial impairment axis of lacrimal gland

The UPR ER contains three upstream stress sensors in the ER membrane, i.e., inositol-requiring enzyme-1, PKR-like ER kinase, and ATF6, which is activated by the presence of unfolded proteins within the ER, ensuring signal transduction to the well-known downstream effector molecules

Yu RH, Wang D, Ren XH, Zeng L, Liu YF