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Targeting the redox-programmed cell death axis in breast cancer: from molecular mechanisms to therapeutic resistance

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introducing how obesity and insulin resistance (initiating factors for the onset of MetS and diabetes) may be opposed by recombinant-human-IGF-1 (rhIGF-1) treatment

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The lower ratio in primed cells could partially suppress TET/JMJ demethylation, helping maintain the higher DNA/histone methylation characteristic of primed pluripotency, whereas the higher ratio in naive cells permits more active demethylation and a hypomethylated state

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