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This review systematically elucidates core pathways through which MPs/NPs drive PD pathology via multidimensional molecular mechanisms: (1) MPs/NPs facilitate -synuclein oligomerization and fibrillation by hydrophobically adsorbing its NAC domain

These data are in line with the previous study reported in vitro for macrophages and human smooth muscle cells, in which -tocopherol inhibits uptake of oxLDL by downregulating CD36 expression (Devaraj et al., 2001)

Antioxidant intake and allergic disease in children

Chemosphere 243, 125362

H., Zhang, H

The mechanism of treatment employs using the intrinsic cellular energy to reduce the active agent lipoate-[S-S] (actually a pro-drug) to dihydrolipoate [DHLA-(SH) 2 ] (the reduced active agent)