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Multiple immune cells through complex interactions accelerates pro-inflammatory and fibrotic processes in the liver (Winer et al., 2016)

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Third, antigenic modulation accelerates AChR internalization and lysosomal degradation through antibody-induced receptor cross-linking, reducing functional receptor density at the neuromuscular junction by approximately 70%90% ( Figure 1 2.1.2 Anti-muscle-specific kinase antibodies Myasthenia gravis mediated by anti-muscle-specific kinase antibodies (MuSK-Ab) exhibits distinct pathogenic mechanisms compared to AChR-MG

Kinesin, a motor protein responsible for anterograde transport of organelles, vesicles, and protein complexes from the cell body to synaptic terminals, causes transport disruption when mutated