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In fibroblast cultures, GHK-Cu stimulates the synthesis of extracellular matrix proteins collagen, elastin and glycosaminoglycans (Maquart et al

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Satizabal, C.L

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The increased traffic in this pathway eventually depletes glutathione reserves, and when glutathione stores fall to 30% of normal, the toxic acetaminophen metabolite can accumulate and promote hepatocellular damage (3)

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